心臟病學英文編修樣本

心臟病學是醫學的一個分科，研究心臟和循環系統的某些部分的疾病，該領域包括先天性心臟病、冠心病、心力衰竭、瓣膜性心臟病和電生理的醫學診斷和治療。Enago在心臟病學領域具有深厚的專業知識，擁有心臟病學和相關學科，如生物儀器、生物材料、生物力學、生物信號處理、生物技術等的專業編輯和校對人員，且已編修了大量此領域相關的科研論文，並協助眾多各國學術作者成功在國際知名SCI期刊上發表高水準論文。

編修後的文稿

For accurate measurement of QT interval measurement, the relationship between QT and the R–R intervals should be reproduciblerepeatable, particularly in cases with. This aissue is important when the heart rate of < 50 bpm and >120 bpm. 1 Moreover, Accurate measurement of the QT interval is also important in athletes and children who have a significant beat-to-beat variability of the R–R interval may need . In such cases, prolonged and numerous recordings for accuracymay be necessary. The longestLongest QT interval is generally observed in the right precordial leads.

Long QT syndrome (LQTS) is a congenital disorder characterized by, which shows a protracted QT interval on the electrocardiogramECG. This condition predisposes patients to the development of This condition influences ventricular tachyarrhythmiatachyarrhythmias to develop in people, which may lead to syncope, cardiac arrest, or sudden cardiac death. AdditionallyIn LQTS, QT prolongation can lead to polymorphic ventricular tachycardia, which is also referred to as torsade de pointes, which in. This condition itself may result inlead to ventricular fibrillation and sudden cardiac death.

Considerably, torsadeTorsade de pointes is widely thought to be triggered by calcium channel reactivation, a delayed sodium current reactivation, or a diminished outward potassium current that results in early afterdepolarization (EAD). This leads to enhanced transmural dispersion of repolarization (TDR) 2 and is usually associated with a prolonged QT interval. TDR serves as a functional reentry background to maintain torsade de pointes. TDR It provides a reentry background for reentry and increases the risk likelihood of EAD, the trigger for torsade de pointes, by extending the extension of the time window for calcium channels to remain open. Any additional further condition that acceleratesing the reactivation of calcium channels reactivation (e.g., increased sympathetic tone), increases the risk of EAD.

Prolonged recovery from excitation increases the probability chance of dispersion of refractoriness dispersion, when some parts of the 3 myocardium are refractory to subsequent depolarization. From a physiological viewpoint, dispersion occurs with repolarization betweenof the three layers of the heart, and the repolarization phase tends to be prolonged in the myocardium. This is the reasonTherefore, why the T wave is usually wide and the interval from the peak of the T -wave to its end (Tp-e) represents the transmural dispersion of repolarization (TDR). In long QT syndrome (LQTS), TDR increases and creates a functional background for transmural reentry.

Editor's Comments

編修後的文稿

For accurate measurement of the QT interval, the relationship between QT and the R–R intervals should be repeatable,. This issue is important particularly when the heart rate is < 50 bpm and >120 bpm. Moreover, aAccurate measurement of the QT interval is also important in athletes and children who have a significant beat-to-beat variability of the R–R interval. In such cases, prolonged and numerous recordings may be necessary. The lLongest QT interval is generally observed in the right precordial leads.

Long QT syndrome (LQTS) is a congenital disorder characterized by, which shows a protracted QT interval on the electrocardiogramECG. LQTS This condition influences ventricular tachyarrhythmias to development in peoplepatients, which may lead to syncope, cardiac arrest, or sudden cardiac death. AdditionallyIn LQTS, QT prolongation can lead to polymorphic ventricular tachycardia, which is also referred to as torsade de pointes. This condition itself may lead to cause ventricular fibrillation and sudden cardiac death.

Torsade de pointes is widely thought to be triggered by calcium channel reactivation, a delayed sodium current reactivation, or a diminished outward potassium current that results in early afterdepolarization (EAD). This leads to enhanced transmural dispersion of repolarization (TDR) 1 and is usually associated with a prolonged QT interval. TDR serves as a functional reentry background to maintain torsade de pointes. ItTDR provides a reentry background for reentry and increases the likelihood of EAD, the trigger for torsade de pointes, by the extension ofextending the time window for calcium channels to remain open. Any additional condition accelerating the reactivation of calcium channels (e.g., increased sympathetic tone), increases the risk of EAD.

Prolonged recovery from excitation increases the probability chance of dispersion of refractoriness, when some parts of the 2 myocardium are refractory to subsequent depolarization. From a physiological viewpoint, dispersion occurs with repolarization of the three layers of the heart, and the repolarization phase tends to be prolonged in the myocardium. This is the reason whyTherefore, the T wave is usually wide and the interval from the peak of the T -wave to its end (Tp-e) represents the transmural dispersion of repolarization (TDR). In long QT syndrome (LQTS), TDR increases and creates a functional background for transmural reentry.

Editor's Comments

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